Macrophage ABHD5 promotes colorectal cancer growth by suppressing spermidine production by SRM

نویسندگان

  • Hongming Miao
  • Juanjuan Ou
  • Yuan Peng
  • Xuan Zhang
  • Yujuan Chen
  • Lijun Hao
  • Ganfeng Xie
  • Zhe Wang
  • Xueli Pang
  • Zhihua Ruan
  • Jianjun Li
  • Liqing Yu
  • Bingzhong Xue
  • Hang Shi
  • Chunmeng Shi
  • Houjie Liang
چکیده

Metabolic reprogramming in stromal cells plays an essential role in regulating tumour growth. The metabolic activities of tumour-associated macrophages (TAMs) in colorectal cancer (CRC) are incompletely characterized. Here, we identify TAM-derived factors and their roles in the development of CRC. We demonstrate that ABHD5, a lipolytic co-activator, is ectopically expressed in CRC-associated macrophages. We demonstrate in vitro and in mouse models that macrophage ABHD5 potentiates growth of CRC cells. Mechanistically, ABHD5 suppresses spermidine synthase (SRM)-dependent spermidine production in macrophages by inhibiting the reactive oxygen species-dependent expression of C/EBPɛ, which activates transcription of the srm gene. Notably, macrophage-specific ABHD5 transgene-induced CRC growth in mice can be prevented by an additional SRM transgene in macrophages. Altogether, our results show that the lipolytic factor ABHD5 suppresses SRM-dependent spermidine production in TAMs and potentiates the growth of CRC. The ABHD5/SRM/spermidine axis in TAMs might represent a potential target for therapy.

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عنوان ژورنال:

دوره 7  شماره 

صفحات  -

تاریخ انتشار 2016